Soft Scrambled Eggs Recipe, Spotlight on Eggs, Vitamin D and Biological Aging, plus Exercise Boosts Brain Health—Even When Energy is Low
There are a few foods that can be satisfying at any meal—breakfast, lunch, or dinner. Whether piled high on toast, topped with a crumble of bacon, or served alongside hearty potatoes, scrambled eggs is one of those dishes. They’re not complicated to make…once your pan is properly prepped. Eggs are a nutrient powerhouse, but there have been lingering concerns about their saturated fat. A study from Australia offers some welcome new thinking. I’m also sharing a study on a very special benefit of vitamin D and a fresh perspective on exercise for brain health.
Soft Scrambled Eggs
Soft Scrambled EggsOne of the simplest dishes can often be the most difficult to make. If your pan isn’t preheated properly or if it isn’t well prepped, scrambled eggs can brown rather than stay yellow, stick in spots, or take seemingly forever to set. Here’s my simple hack for eggy perfection.
Ingredients
- 4 extra-large eggs
- 2 tablespoons extra virgin olive oil, plus more for drizzling
- Fine sea salt
- Freshly cracked black pepper
Directions
Step 1
Heat your sauté or fry pan over moderately high heat for 5 minutes. Meanwhile, thoroughly whisk the eggs in a large bowl until no whites are visible.
Step 2
When the pan is ready (a drop of water will sizzle and evaporate), lower the heat to medium-high and add the olive oil, rotating the pan so that it covers the bottom surface. Pour the eggs into the pan and immediately start moving them around with a heat-resistant spatula so that they cook evenly. Constant movement will help prevent brown spots. Take the eggs off the heat just before they’re completely firm—they will keep cooking.
Step 3
Divide the eggs between two plates, season with salt and pepper, and drizzle with olive oil.
Serves 2
Healthy Ingredient Spotlight
A Fresh Look at Eggs
Chances are you’ve read conflicting advice over the years about whether to eat eggs and how many per day or week to have. A new study from Monash University in Melbourne, Australia, found that eggs have certain benefits for a specific part of the population.
Published in the journal Nutrients, the study found that for relatively healthy older adults, consuming eggs 1 to 6 times per week was associated with a lower risk of all-cause mortality (death from any cause) and CVD mortality compared to those who rarely or never eat eggs.
The study involved 8,756 adults aged 70 years or older, who self-reported the frequency of their total egg intake as never/infrequently (1 to 2 times/month), weekly (1 to 6 times/week), or daily (daily/several times per day), as part of the ASPREE Longitudinal Study of Older Persons (ALSOP) sub-study. ASPREE (ASPirin in Reducing Events in the Elderly) was a large primary prevention aspirin trial of more than 19,000 participants in Australia and the US, mostly 70 years and older, and continues today as a longitudinal study.
First author Holly Wild, a PhD candidate and lecturer from the Monash University School of Public Health and Preventive Medicine, said that, compared to older adults who never or infrequently ate eggs, those who ate eggs 1 to 6 times a week had a 15 percent lower risk of death from any cause and a 29 percent lower risk of cardiovascular disease-related death. “Eggs are a nutrient-dense food, a rich source of protein, and a good source of essential nutrients, such as B vitamins, folate, unsaturated fatty acids, fat-soluble vitamins (E, D, A, and K), choline, and numerous minerals and trace elements,” Wild said. “Eggs are also an accessible source of protein and nutrition in older adults, with research suggesting that they are the preferred source of protein for older adults who might be experiencing age-related physical and sensory decline.”
The work also explored the relationship between egg consumption and mortality across different levels of diet quality—low, moderate, and high. “The study found that older adults with a moderate- to high-diet quality reported a 33 percent and 44 percent lower risk of CVD-related death, suggesting that the addition of eggs to moderate- and high-quality diets may improve longevity,” the researchers wrote.
The current Australian Dietary Guidelines and the American Heart Association (AHA) recommend that adults with normal cholesterol can eat up to seven eggs per week, while some European countries suggest limiting eggs to 3 to 4 per week. The AHA also supports up to 2 eggs per day for older adults with normal cholesterol.
“Previous research has observed a higher risk of mortality with egg consumption for those who have high cholesterol. For this reason, we also explored the association between egg consumption and mortality in people with and without dyslipidemia (clinically diagnosed high cholesterol),” Wild said. “We found a 27 percent lower risk of CVD-related death for participants with dyslipidemia who consumed eggs weekly, compared to their counterparts that consumed eggs rarely or never, suggesting that in this study cohort, the presence of dyslipidemia does not influence the risk associated with egg consumption. Our results suggest that eating up to six eggs a week may reduce the risk of death from all causes and cardiovascular-related diseases in older adults. These findings may be beneficial in the development of evidence-based dietary guidelines for older adults.”
Of course, everyone is unique. Before increasing how many eggs you eat, talk to your doctor to see what makes sense for your health profile.
Quick Kitchen Nugget
Eggs: Checking for Freshness
Want a quick hack to see whether the eggs in your fridge are still fresh? Try the float test. Fill a large bowl or even a wide glass with cold water and gently add an egg. If it sinks and rests on one side, it’s fresh. If it sinks but stands upright, use it right away. If it floats, toss it. An egg that floats has lost moisture through the shell and its contents have shrunk.
For Your Best Health
Vitamin D and Biological Aging
Results of a landmark study called the VITAL randomized controlled trial reveal that vitamin D supplementation helps maintain telomeres, the protective caps at the ends of chromosomes that shorten during aging and are linked to the development of certain diseases. The report, published inThe American Journal of Clinical Nutrition, is based on data from the VITAL Telomere sub-study co-led by researchers at Mass General Brigham and the Medical College of Georgia, and supports a promising role in slowing a pathway for biological aging.
VITAL looked at the effects of vitamin D3 (2,000 IU/day) and omega 3 fatty acid (1 g/day) supplementation on US women aged 55 years and older and men aged 50 years and older for five years. The sub-study included 1,054 participants, whose telomere length in white blood cells was assessed at baseline and at year 2 and year 4.
“VITAL is the first large-scale and long-term randomized trial to show that vitamin D supplements protect telomeres and preserve telomere length,” said co-author JoAnn Manson, MD, principal investigator of VITAL and chief of the Division of Preventive Medicine at Brigham and Women’s Hospital, a founding member of the Mass General Brigham healthcare system. “This is of particular interest because VITAL had also shown benefits of vitamin D in reducing inflammation and lowering risks of selected chronic diseases of aging, such as advanced cancer and autoimmune disease.”
Telomeres are made of repeating sequences of DNA, or base pairs, that prevent chromosome ends from degrading or fusing with other chromosomes. Telomere shortening is a natural part of aging and is associated with an increased risk of various age-related diseases.
A few short-term small-scale studies have suggested that vitamin D or omega 3 fatty acid supplementation may help support telomeres, but results have been inconsistent. This study showed that compared with taking a placebo, taking vitamin D3 supplements significantly reduced telomere shortening over four years, preventing the equivalent of nearly three years of aging compared with placebo. Omega 3 fatty acid supplementation had no significant effect on telomere length throughout follow-up.
“Our findings suggest that targeted vitamin D supplementation may be a promising strategy to counter a biological aging process, although further research is warranted,” said Haidong Zhu, PhD, first author of the report and a molecular geneticist at the Medical College of Georgia, Augusta University.
Fitness Flash
Exercise Boosts Brain Health—Even When Energy is Low
We know exercise is good for our body, but what about our brain? A new study from the University of Missouri suggests that exercise plays a crucial role in keeping our mind sharp, even when one of the brain’s key energy sources isn’t available. The study, led by researchers Taylor Kelty, PhD, and R. Scott Rector, PhD, offers fresh insight into brain health and suggests that exercise could play a bigger role in preventing cognitive decline than previously thought.
When the body runs low on its usual fuel—glucose—the liver makes ketones, an alternative energy source that helps power the brain. These molecules support cognitive memory, learning, and overall brain health. But what happens when the liver can’t produce enough of them? Can exercise help compensate for this ketone deficiency? Dr. Rector, the director of the Roy Blunt NextGen Precision Health building and a professor in the School of Medicine, and Dr. Kelty were determined to find out.
For the study, they looked at what happens when ketone production is limited in the liver. As expected, the result was a noticeable decline in brain function and memory. But then something remarkable happened: Exercise managed to help reverse some of the cognitive decline, even with the ketone production impaired.
“Going into the study, we thought that with fewer ketones and the cognitive impairments that causes, exercise may not be able to overcome that impairment,” said Dr. Kelty, a postdoctoral fellow in Dr. Rector’s lab. “But it seems like exercise is so powerful that there are other mechanisms going on in the brain that allow it to circumvent those impairments and still receive the benefits from exercise.”
“This study highlights how exercise benefits the body in a multitude of ways, even when we don’t fully understand all the molecular mechanisms involved,” Dr. Rector said. “Even when we remove a single pathway, exercise is doing so many other things that it can help mitigate those deficiencies.”
The findings are especially promising for people with liver conditions that prevent the body from making ketones. Dr. Kelty pointed out that the emerging field of liver-brain research is beginning to show that individuals with severe liver dysfunction have a higher risk of developing dementia. “If ketone production in the liver is disrupted, it could be a potential cause of cognitive decline, ultimately leading to conditions like dementia,” he said.
Ideally, this research helps raise awareness about the importance of ketone production to brain health and the impact exercise can have on staying mentally sharp. “There’s so much we’re still uncovering, and with all the state-of-the-art resources and interdisciplinary collaborations at Mizzou, it’s exciting to think about where this research might take us next,” Dr. Kelty said. “Exercise could be a key piece of the puzzle in preserving brain health as we age.”
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